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Phosphorylation at S87 is enhanced in synucleinopathies inhibits a-synuclein oligomerization and influences synuclein-membrane interactions

(2006) demonstrated consistent external currents in human skin and rodent corneal and skin wounds, suggesting that phosphatidylinositol-3-OH kinase-g (PI(3)Kg) and phosphatases as well as tensin homologues (PTEN) control cellular orientated migration and identified the first gene that regulates cell motility and wound currents to promote wound healing

10.1093/hmg/ddt319 388 WangW.WangL.LuJ.SiedlakS

et al., 2020b)

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